17 Numerous molecules may induce the retinal vascular hyperpermeability that leads to macular edema. Post-mortem detection of six human herpesviruses (HSV-1, HSV-2, VZV, EBV, CMV, HHV-6) in trigeminal and facial nerve ganglia by PCR.
Herpes zoster-associated encephalitis: clinicopathologic report of 12 cases and review of the literature.
Unilateral acute uveitis with retinal periarteritis and detachment.
Herpes zoster ophthalmicus following bone marrow transplantation in children. Moesen I, Khemka S, Ayliffe W. Acute retinal necrosis secondary to herpes simplex virus type 2 with preexisting chorioretinal scarring. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvMTIyMzA0Ny1jbGluaWNhbA== Peck R, Gimple SK, Gregory DW, Youree B.
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Sharma A, Makrandi S, Modi M, Sharma A, Marfatia Y. Neves RA, Rodriguez A, Power WJ, Muccioli C, Lane L, Belfort R Jr, et al. Herpes zoster and postherpetic neuralgia: prevention and management. Intravenous foscarnet in the management of acyclovir-resistant herpes simplex virus type 2 in acute retinal necrosis in children. The visual outcome of eyes affected by ARN is variable and largely depends upon vision at time of presentation as well as whether or not secondary retinal detachment or ischemic optic neuropathy occurs.
Genetics are believed to play a role in an individual’s risk for ARN with antigen expression of: Acute retinal necrosis: clinical features, early vitrectomy, and outcomes.
Intrathecal methylprednisolone for intractable postherpetic neuralgia.
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Acyclovir with and without prednisone for the treatment of herpes zoster. Acute retinal necrosis (ARN) is an inflammatory condition which may present as panuveitis. Recommendations for the management of herpes zoster. Chiriac A, Chiriac AE, Pinteala T, Moldovan C, Stolnicu S. Allergic Contact Dermatitis from Topical Acyclovir: Case Series.
Retinal laser photocoagulation was then applied to the border of the white-yellow patched lesions to prevent the late consequence of retinal … Herpes zoster in ophthalmic (V1) distribution of trigeminal nerve. Holland GN. Svozílková P, Ríhová E, Diblík P, Kuthan P, Kovarík Z, Kalvodová B. Varicella zoster virus acute retinal necrosis following eye contusion: case report.
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Cystoid macular edema (CME) is a form of macular retina thickening that is characterized by the appearance of cystic fluid-filled intraretinal spaces. It has classically been diagnosed upon investigation after a decrease in visual acuity; however, improvements in imaging technology make it possible to noninvasively detect CME even before a clinically significant decrease in central vision. The commencement of second eye infection ranged from 24 hours to 6 months later.Occlusive retinal vasculitis involving arteries and veinsOne or more focus of retinitis, resulting in necrosis with discrete borders located in the retinal periphery with circumferential spreadMost cases of acute retinal necrosis have been reported to be caused by the followingCareful and complete ophthalmological examination should be undertaken in all patients with anterior uveitis.